— ENDOGENOUS ACTIN-SEQUESTERING PEPTIDE · WOUND & TISSUE REPAIR SIGNAL

TB-4.

An endogenous 43-amino-acid peptide found at high concentrations in virtually all human cells. Thymosin Beta-4 is the body's primary actin-sequestering protein — controlling the cytoskeleton dynamics that drive wound healing, cardiac repair, and tissue regeneration.

Key Signals
Wound · Cardiac · Repair
Origin
Endogenous (all human cells)
Investigator
Low et al., 1981
Class
Actin-sequestering peptide (43 AA)
Status (UAE)
Research compound (off-label)
— THE RESEARCH STORY

The Repair Peptide Hidden in Plain Sight

Low et al. isolate Thymosin Beta-4 from bovine thymus in 1981; later found at high levels in platelets and wound fluid.

Identified as the primary G-actin sequesterer in human cells — controlling cytoskeletal dynamics critical for wound closure.

RxBio advances TB-4 into Phase I/II trials for wound healing and dry eye syndrome — the first actin peptide in human trials.

— WOUND CLOSURE RESPONSE
CONCEPTUAL · RODENT EXCISIONAL MODEL
25% 50% 75% 100% DAY 0 DAY 3 DAY 7 DAY 14 DAY 21
TB-4 TREATED UNTREATED CONTROL
— THE MOLECULE

The body's own repair blueprint

PRIMARY STRUCTURE

Ser-Asp-Lys-Pro-Asp-Met-Ala-Glu-Ile-Glu-Lys
Phe-Asp-Lys-Ser-Lys-Leu-Lys-Lys-Glu-Thr-Gly
Gln-Glu-Lys-His-Ser-Glu-Gly-Ala-Ile-Ile-Lys
Lys-Val-Ile-Glu-Glu-Glu-Leu-Lys-Pro-Leu

C₂₁₂H₃₅₀N₅₆O₇₈S · MW 4964 Da

4964MOLECULAR WEIGHT Da
43AMINO ACIDS
Ph.IICLINICAL STAGE

A 43-residue peptide present at high concentrations in platelets, white blood cells, and wound fluid — binding G-actin with high affinity and acting as master regulator of actin dynamics across human cell types.

— AN HONEST READING OF THE DATA

Already in you — now studied.

Preclinical signal is unusually robust; human trial data exists but remains limited in scale.

TIER 01 / PRECLINICAL
Robust Animal Evidence
  • Wound closure (excisional)
  • Cardiac repair (post-MI)
  • Corneal wound healing
  • Hair follicle activation
  • Anti-inflammatory signal
TIER 02 / HUMAN TRIALS
Phase I–II Level Data
  • Wound healing (Phase II)
  • Dry eye syndrome (Ph.II)
  • Safety confirmed (Ph.I)
  • No approved indication
TIER 03 / SPECULATIVE
Extrapolated & Unverified
  • Neurological regeneration
  • Systemic anti-aging use
  • Muscle growth claims
  • Spinal cord repair
— TB-4 MECHANISMS & RESEARCH DOSING READY

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Mechanism deep-dive and research dosing protocols — available to registered researchers. It's free and takes under a minute.

— REGULATORY LANDSCAPE

TB-4's Closest Approach to Approval

In the UAE, research-grade peptides occupy a defined regulatory category distinct from licensed medicines. Compounds sold under this classification — including those offered by Dubai Peptides — are intended exclusively for qualified research applications, not for human administration.

TB-4 is unique among research peptides in having completed Phase I safety trials and a Phase II study for neurotrophic dry eye syndrome — the most advanced actin-binding peptide to reach human trials. No new drug application has been filed despite positive safety data; commercial development stalled after the Phase II endpoint. Being endogenous, TB-4 is present in every human cell — a rare distinction.

1981 / ISOLATION

Low et al. isolate Thymosin Beta-4 from bovine thymus; found in all human cells at high levels.

2000s / CLINICAL TRIALS

RxBio initiates Phase I/II wound-healing and dry eye studies — first actin peptide in trials.

PRESENT / UAE MARKET

An off-label compounding market for TB-4 exists in the UAE, operating outside the research-use classification this platform follows.